ABSTRACT
This article has been retracted at the authors' request.
ABSTRACT
Eosinophilic enteritis is an uncommon disease of unknown cause characterized by eosinophilic infiltration in various areas of the gastrointestinal tract with symptoms. It is generally classified according to the layer of the gastrointestinal tract involved. Eosinophilic infiltration of the serosa is the rarest form of presentation and may manifest eosinophilic ascites. We report a case of a 47-year-old man who experienced progressing abdominal pain. A diffuse erythematous change of the gastric mucosa was observed on gastrofibroscopy. An abdominal computed tomography and colonoscopy showed diffuse wall thickening of the small bowel and colon with a small amount of ascites. Eosinophilic infiltration was confirmed by multiple biopsies of the gastrointestinal tract and peritoneal fluid analysis. The patient was treated with corticosteroid and responded dramatically.
Subject(s)
Humans , Middle Aged , Abdominal Pain , Ascites , Ascitic Fluid , Biopsy , Colon , Colonoscopy , Enteritis , Eosinophilia , Eosinophils , Gastric Mucosa , Gastrointestinal Tract , Serous MembraneABSTRACT
Eosinophilic enteritis is an uncommon disease of unknown cause characterized by eosinophilic infiltration in various areas of the gastrointestinal tract with symptoms. It is generally classified according to the layer of the gastrointestinal tract involved. Eosinophilic infiltration of the serosa is the rarest form of presentation and may manifest eosinophilic ascites. We report a case of a 47-year-old man who experienced progressing abdominal pain. A diffuse erythematous change of the gastric mucosa was observed on gastrofibroscopy. An abdominal computed tomography and colonoscopy showed diffuse wall thickening of the small bowel and colon with a small amount of ascites. Eosinophilic infiltration was confirmed by multiple biopsies of the gastrointestinal tract and peritoneal fluid analysis. The patient was treated with corticosteroid and responded dramatically.
Subject(s)
Humans , Middle Aged , Abdominal Pain , Ascites , Ascitic Fluid , Biopsy , Colon , Colonoscopy , Enteritis , Eosinophilia , Eosinophils , Gastric Mucosa , Gastrointestinal Tract , Serous MembraneABSTRACT
PURPOSE: To introduce mini-open rotator cuff repair using the anterolateral approach and evaluate its clinical outcomes and effectiveness. MATERIALS AND METHODS: 59 consecutive cases of rotator cuff tearing which were treated with mini-open repair utilizing the anterolateral approach were evaluated. The population comprised 39 men and 20 women, with an average age of 56.6 years. An average follow-up time period was 26 months. Clinical outcomes were analyzed based on VAS, ADL, and ASES scores. RESULTS: The average respective VAS, ADL, and ASES scores improved from 7.04, 12.37, and 35.32 preoperatively to 1.02, 27.20, and 90.08 postoperatively (p=0.000). There were 41 excellent, 11 good, 2 fair, and 5 poor results. There were satisfactory results in 52 cases (88.1%). There were no statistically significant differences between the final ASES scores and age, sex, duration of symptoms, tear size, and preoperative stiffness (p>0.05). CONCLUSION: Mini-open rotator cuff repair using the anterolateral approach effective in providing better visualization.
Subject(s)
Female , Humans , Male , Activities of Daily Living , Follow-Up Studies , Rotator CuffABSTRACT
If ventricular fibrillation develops after acute myocardial infarction, it should be treated immediately with the external electrical cardioversion and antiarrhythmic agents, followed by the rapid correction of the reversible and underlying cause. Nevertheless, if ventricular fibrillation recurs, ICD(implantable cardioverter-defibrillator) therapy is necessary. ICD is an important nonpharmacological option in the treatment of malignant ventricular arrhythmias. A 53 year old man was admitted into our hospital with a severe chest pain of 9 days. On cardiac catheterization, a significant stenosis at the mid-left anterior descending coronary artery and a huge ventricular aneurysm were found. Then PTCA(percutaneous transluminal coronary angioplasty) with a stent was taken. Three days later, two episodes of ventricular fibrillation developed and it was treated with electrical cardioversion and antiarrhythmic agents. On the second look of cardiac catheterization, a subacute closure of the stented site was detected and then the culprit leision was recanalized with repeated PTCA and another stent was implanted. But ventricular fibrillation recurred although the stented lesion still patent on the third look of cardiac catheterization. After all, we implanted ICD on him. 46 Episodes of ventricular fibrillations developed at the same day and the next day of ICD implantation. All of the episodes of ventricular fibrillation were successfully converted to sinus rhythm with shocks from ICD. Since then, the frequency of ventricular fibrillation decreased with IABP(intraaortic balloon pump) therapy. We report the first case of malignant ventricular fibrillation after myocardial infarction which was uncontrolled with conventional therapy, but effectively treated with ICD.